DYNAMIC
BRACING AND IT'S EFFECT ON THE KNEE
by Gary R. Bledsoe, President/CEO - Bledsoe Brace Systems
Educational Series - CP030014
Dynamic bracing is the
use of muscle power to cause a brace to work against the pathological movement of a joint.
In the case of the knee, different pathological movements are possible depending on which
stabilizing structures are injured. It is necessary to determine which structures are
damaged, then to discover at what point in the range of motion these abnormal movements
occur. This information permits designing an orthosis that uses leg extension or flexion
to precompress soft tissues with increasing force to prevent pathological motion as the
leg approaches the range where instability occurs. Good examples of pathological
conditions which can be addressed with this type of brace are found in ACL deficient knees
and knees with medial or lateral gonarthrosis.
The anterior cruciate
ligament (ACL) is the most often injured ligament in the knee. The ACL can be torn in
young athletes at a force of 33N per Kg of body mass, or 3.3 times body weight.1
Even at such high force levels, 80% of the knees are injured with no external contact.2
In the absence of the
ACL, the quadriceps muscles can sublux the tibia in the final range of extension.3-7
The quadriceps muscle may also be inhibited by this subluxation.8,9 This
inhibition coupled with the immobilization that occurs after injury is believed to cause
quadriceps atrophy.7,10,11 To further complicate matters, the hamstrings
reaction time may be greatly slowed, and increased co-contraction is typically present.8
Symptomatic patients have been observed with changes in force and muscle pattern
activation in activities as simple as level walking.12 The rehabilitation of
these injuries is time consuming for the average patient. It requires long periods of
physical therapy with hamstrings training and a great deal of work to overcome quadriceps
atrophy.13-17
Most ACL deficient
patients that are symptomatic have great difficulty in performing maneuvers such as
stopping, landing from a jump, running downhill, or making rapid lateral maneuvers. In
each of these maneuvers, rapid open kinetic chain extension of the leg, coupled with a
greater degree of extension, occurs a brief instant before foot strike thus permitting
free subluxation and subsequent sensations of instability or even "giving way"
of the knee.9
The knee is not
just a mechanical structure. It is also a neurological structure that is filled with four
types of mechano-receptor nerves.18-21 Each type of nerve functions at a
different force threshold, and the various force and movement conditions are compiled to
form a kinesthetic awareness that we call proprioception.22-30 Slower movements
permit direct feedback of information to control muscle function, joint position, and
stability, while more rapid movements that are too fast to permit direct control cause
changes in muscle and force patterns.31
ACL deficient
knees have been shown to have reduced proprioception compared to normal knees.32
The loss of proprioception is believed to be caused by the loss of the mechanoreceptors in
the missing ACL.33,34 A part of proprioception that seems to be particularly
affected is joint position sense.25 Newberg found a significant difference in
joint position sense between the operative and opposite knee up to 15 months post-op,
while Barrack et al found it to be normal after 31 months.32,35 This may
explain why reconstructed knees may not "feel" normal to the patient for up to
two years. Other researchers have found that muscle training, bracing, and the application
of elastic bandages improves joint position sense.36-38
Functional bracing is
intended to reduce or eliminate the instability of the knee following ligamentous injury.39,40
It is currently indicated for frequent episodes of "giving way", poor quadriceps
and hamstrings strength, high ligamentous laxity, and the desire to return to competitive
sports.39 Patients who wore functional braces reported fewer episodes of giving
way, decreased pain and swelling, increased confidence, and an increased ability to return
to their former level of activity.41-45 The stability and comfort of functional
braces depends on many factors including the design and proper placement of the brace.46
To better understand brace design, it is first necessary to understand the factors that
limit and enhance control of the leg.

The greatest factor
that limits control of abnormal movements by any external support device, including
braces, is soft tissue movement and compression. Figure A shows a typical soft tissue
compression curve of a 5cm wide strap on the posterior hamstrings. Using traditional
orthotic principles, optimization of control involves increasing brace length to increase
leverage, and increasing surface contact area of the straps and shells to reduce tissue
compression for a given force level (Fig B). There is an upper limit to this approach in
terms of the

permissible placement
of straps and shells to achieve movement of the leg. Figure C shows that it is possible to
achieve 20 mm of tibial subluxation with only 10 mm of hamstrings compression using
typical design limitations. It is very easy to achieve this amount of compression during
leg extension because the hamstrings are acting only as antagonists. This explains why
traditional orthoses have difficulty in limiting tibial movement at forces experienced
during sports play.


Braces with no net
shear force can be termed passive braces (Fig D).

Large subluxations are possible before soft tissue is compressed enough to provide high
resistance (Fig E).
It is possible in some
brace designs to adjust strap tension to precompress soft tissue by placing a preloaded
shear force against the knee. These braces can be termed static

counter force braces (Fig F). Although some increase in control force is possible,
circulation places a limit on the amount of

preload possible in a
static tensioned strap (Fig G).
Dynamic bracing uses
the act of leg extension to place an increasing shear force across the knee as the leg is
extended (Fig H). This effectively precompresses the soft tissues to


very high levels dependent on design and patient tolerance (Fig I). The force is reduced
as the leg is flexed so that circulation is not inhibited during sports play. This
movement against the knee is readily apparent when the brace is seen in motion. Many
braces may lack mechanical effectiveness but enhance the ability to sense abnormal
movements in a symptomatic knee.
Dynamic braces provide
varying compressive forces against the leg that the patient can correlate with angle to
enhance joint position sense and restore quadriceps function in symptomatic knees. In two
recent studies, Acierno et al 46,47, demonstrated the effect of dynamic braces
on normal and ACL deficient knees using EMG during maximal effort isokinetic extension. It
was shown that asymptomatic knees apparently compensate for the absence of the ACL and do
not therefore exhibit quadriceps inhibition. The study also shows the effectiveness of a
dynamic brace in stabilizing the tibia against subluxation thus reducing the
hamstrings contraction, and permitting an increase in extension torque in
symptomatic knees.47


Dynamic bracing is not
just limited to the ACL deficient knee. It can be used on the reconstructed ACL to reduce
stress on the reconstruction and permit earlier mobilization and quadriceps exercise46.
The same principles can be used to reduce posterior subluxation for posterior cruciate
problems by placing an anterior load on the tibia during knee flexion.
It is possible to use
the principles of dynamic bracing to place varus or valgus loads on the knee for problems
such as medial and lateral collateral ligaments, tibial plateau fractures, and
osteoarthritis. In gonarthrosis it is particularly effective. When a dynamic brace is seen
extending on the leg, it is easy to see the brace placing a large valgus load on a knee
with medial gonarthrosis. As the leg is extended, the brace uses muscle power to
dynamically close the lateral compartment and open the medial compartment of a varus knee
with osteoarthritis to control pain.


In a recent preliminary
study by Noyes et al49, patients that used the Bledsoe Thruster MA Brace for
only 3 weeks were able to increase their walking time by a factor of five. They increased
their function grade and activity level by 20%- 25%.

At the same time, their average pain level during this increased activity and increased
walking time was reduced from an average analog score of 7 to less than 3 (Fig J, K, L).49
Most of the patients were able to completely eliminate the need for pain and
anti-inflammatory medications. The brace is very helpful in delaying the need for high
tibial osteotomy or total knee replacement. The brace can also be used to test the
effectiveness of an HTO, or to help hold the HTO closed after surgery thus permitting
earlier motion and ambulation.
Summary
Dynamic bracing is
capable of overcoming many of the obstacles that limit traditional passive orthoses, but
it is not a cure-all. It is an interesting new tool that can be optimized to provide
higher forces to control certain pathological conditions that are not possible to control
with conventional designs. It expands the control that a brace can provide. There are many
interesting possibilities as we search for ways to use these new principles for other
joints and other pathological conditions.
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